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CHE Partnership call: Using Science to Set Regulatory Criteria: Identifying Endocrine Disrupting Chemicals in the European Union
Wed, June 29
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CHE Partnership call: Interactive Effects of Multiple Pesticides on Human Health – A 2016 California Report
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CHE Partnership call: A Consensus on the Environmental Chemicals Contributing to Neurodevelopmental Disorders: Project TENDR
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CHE Partnership call: Autism Pathways Analysis: A Functional Framework and Clues for Further Investigation
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CHE Partnership call: Fatty Bones Make Bad Skeletons: Influence of Bone-disrupting Chemicals across the Lifespan
Tues, July 26
 

6/22/16: Clarifying EMF and Cancer: Precautionary Occupational Strategies and Results of the NTP Cell Phone Studies
This call was not recorded


6/15/16: MP3 recording available: Mercury in the North: Sources of Contamination and International Policies to Protect Health & Human Rights

 

6/7/16: MP3 recording available: Exploring Multifactorial Contributors to Disease Outcomes: The Possible Role of Acetaminophen in Asthma and Autism


5/24/16: MP3 recording available: The Human Microbiome and Health Effects on Prenatal Microbiome Exposure

 

5/18/16: MP3 recording available: Signaling Mechanisms by Which Xenoestrogen Pollutants Disrupt Normal Estrogenic Signaling


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CHE Partners on why they value our work

Gut Microbiota and Environmental Chemicals in Diabetes and Obesity

Jan 19, 2012

Exposure to environmental chemicals has been associated with the development of diabetes and obesity in numerous epidemiological and animal studies. Gut microbiota, the microorganisms that colonize the intestine, also appear to play a role in diabetes and obesity. Do these two environmental factors interact? Our speakers discussed evidence that suggests that they may.

Dr. Jacobs has published numerous articles on exposure to persistent organic pollutants in relation to health, including diabetes and obesity. In a recent study, he and colleagues investigated the link between methanogen microbes, persistent organic pollutants (POPs), and obesity. These microbes biodegrade petroleum hydrocarbons in polluted environments, and the authors hypothesized that a higher body burden of petroleum based chemicals may promote methanogens in the gut. The results show that the levels of methangens were associated with measurements of obesity, as well as blood POP levels.

In a recent review, Dr. Snedeker and Dr. Hay integrate the evidence linking gut microbiota and environmental chemical exposures to diabetes and obesity, providing a framework for how these factors may interact in these diseases, and identifying future research needs. Gut microbiota may affect the absorption, distribution, metabolism, and excretion of environmental chemicals. Variations in gut microbiota are likely to affect an individual's exposure to obesogenic and diabetogenic chemicals.

Featured included:

David Jacobs, PhD, Division of Epidemiology and Community Health, School of Public Health, University of Minnesota.

Suzanne Snedeker, PhD, Department of Food Science, Cornell University.

Anthony Hay, PhD, Department of Microbiology, Institute for Comparative and Environmental Toxicology, Cornell University.

The call was moderated by Steve Heilig, MPH, CHE Director of Public Health & Education, and Director of Public Health & Education, San Francisco Medical Society.                                                                            

 

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